Statin Risks

The full range of statin side effects includes cognitive dysfunction, behavioral and emotional disorders, chronic nerve and muscle damage and an ALS-like neuromuscular degenerative process, as major categories of damage.

Thousands of statin users have been afflicted with peripheral neuropathies with a tendency to be resistant to all traditional medical treatment.

Statins inhibit not only dolichols, corrupting our DNA damage correction, but CoQ10 as well, increasing our damage load. Predictably the inevitable effect is increased mitochondrial DNA damage — considered by many authorities to be the mechanism of our aging process as well as that of many chronic diseases.

Many of the statin side effects are permanent and weakness and fatigue are common complaints. Many statin victims say that abruptly, almost in the blink of an eye, they have become old people.

Statins block the synthesis of CoQ10 and dolichols, thereby contributing directly to the premature common chronic ills of aging. Since this involves normal physiologic processes, it is silent. By the time we become aware of it, it is already far too late and the damage has been done to those susceptible.

The above is from a description of the book, The Dark Side of Statins plus the Wonder of Cholesterol , by Dr. Duane Graveline. The final chapter is a first-hand account of Dr Graveline’s last weeks and days and the official causes of his death that he attributes directly to statins.


This video will start at 10:48 – How the drug companies succeeded in acquiring new customers.
Stents & Statins – Do they work? A top cardiologist’s view


  1. How Statin Drugs Really Lower Cholesterol: And Kill You One Cell at a Time
  2. The Great Cholesterol Myth
  3. The Dark Side of Statins plus the Wonder of Cholesterol
  4. Fat and Cholesterol Don’t Cause Heart Attacks and Statins are Not The Solution
    This book is dedicated to Uffe Ravnskov, MD, Ph.D. for his seminal and propaedeutic achievements in disputing the dogma that fat and cholesterol cause coronary heart disease, and that statins are safe and cardioprotective for everyone. As will be seen, no studies support the notion that restricting fat reduces coronary morbidity or mortality. More importantly, government recommendations mandating low fat diets are likely the cause of the escalating epidemic of obesity and type 2 diabetes. Several chapters detail the panoply of significant adverse health effects of statins that have been ignored or suppressed in reports of drug company sponsored trials. These include promoting the development of coronary atherosclerosis and congestive failure. In addition, the putative benefits of statins are clearly unrelated to lowering LDL or cholesterol, but rather anti-inflammatory and especially anticoagulant activities. This clotting or “atherothrombotic” hypothesis appears to explain all of the factors known to cause or protect against coronary heart disease. Other chapters by THINCS members discuss the role of infections and sulfur deficiency, and the numerous ways data are doctored to hype the benefits and minimize the dangers of statins. All of these contributions expose the fallacies of the lipid hypothesis, which was called “the greatest scientific deception of this century, perhaps of any century” by the distinguished nutritionist George Mann, former Co-Director of the Framingham Study.

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